Visfatin regulates Pg LPS-induced proinflammatory/prodegradative effects in healthy and inflammatory periodontal cells partially via NF-κB pathway - ScienceDirect
Chlorogenic Acid as a Positive Regulator in LPS-PG-Induced Inflammation via TLR4/MyD88-Mediated NF-κB and PI3K/MAPK Signaling Cascades in Human Gingival Fibroblasts
Role of TLR4 signaling on Porphyromonas gingivalis LPS-induced cardiac dysfunction in mice | PLOS ONE
In Vivo Up-Regulation of Kinin B1 Receptors after Treatment with Porphyromonas gingivalis Lipopolysaccharide in Rat Paw | Journal of Pharmacology and Experimental Therapeutics
GI Bacteria Affects Rheumatoid Arthritis Disease Markers | Chondrex, Inc.
Inflammatory signals in human oral keratinocytes triggered by P.... | Download Scientific Diagram
Pg ant its LPS increase ECs death.
Porphyromonas gingivalis lipopolysaccharides act exclusively through TLR4 with a resilience between mouse and human | Scientific Reports
The promotion of nephropathy by Porphyromonas gingivalis lipopolysaccharide via toll-like receptors | Diabetology & Metabolic Syndrome | Full Text
The Effect of Porphyromonas gingivalis Lipopolysaccharide on the Pyroptosis of Gingival Fibroblasts | SpringerLink
Frontiers | Pathogenesis of Important Virulence Factors of Porphyromonas gingivalis via Toll-Like Receptors
IJMS | Free Full-Text | P. gingivalis-LPS Induces Mitochondrial Dysfunction Mediated by Neuroinflammation through Oxidative Stress
Chlorogenic Acid as a Positive Regulator in LPS-PG-Induced Inflammation via TLR4/MyD88-Mediated NF-κB and PI3K/MAPK Signaling Cascades in Human Gingival Fibroblasts
IJMS | Free Full-Text | Porphyromonas gingivalis Lipopolysaccharides Promote Proliferation and Migration of Human Vascular Smooth Muscle Cells through the MAPK/TLR4 Pathway
![Porphyromonas gingivalis lipopolysaccharide promotes T-hel per17 cell differentiation by upregulating Delta-like ligand 4 expression on CD14+ monocytes [PeerJ]](https://dfzljdn9uc3pi.cloudfront.net/2021/11094/1/fig-1-full.png "Porphyromonas gingivalis lipopolysaccharide promotes T-hel per17 cell differentiation by upregulating Delta-like ligand 4 expression on CD14+ monocytes [PeerJ]")
Porphyromonas gingivalis lipopolysaccharide promotes T-hel per17 cell differentiation by upregulating Delta-like ligand 4 expression on CD14+ monocytes [PeerJ]
Porphyromonas gingivalis Stimulates Bone Resorption by Enhancing RANKL (Receptor Activator of NF-κB Ligand) through Activatio
Circular RNA PPP1CC promotes Porphyromonas gingivalis-lipopolysaccharide-induced pyroptosis of vascular smooth muscle cells by activating the HMGB1/TLR9/AIM2 pathway - Jie Liu, Yong Wang, Yaoyun Liao, Ying Zhou, Jijin Zhu, 2021
Frontiers | Pathogenesis of Important Virulence Factors of Porphyromonas gingivalis via Toll-Like Receptors
Anti-P.gingivalis mAb (PG antibody)（Monoclonal, AW1-4） | Antibody | MBL Life Sience -ASIA-
Porphyromonas Gingivalis in the Pathogenesis of Alzheimer's Disease and Its Therapeutic Target
Molecules | Free Full-Text | Porphyromonas gingivalis Lipopolysaccharide Induced Proliferation and Activation of Natural Killer Cells in Vivo
Porphyromonas gingivalis lipopolysaccharide increases lipid accumulation by affecting CD36 and ATP-binding cassette transporter A1 in macrophages
P.g.-infection and LPS-PG induce myofibroblastic activation of HSCs.... | Download Scientific Diagram
em>Porphyromonas gingivalis</em> lipopolysaccharide enhances the proliferation of human periodontal ligament cells via upregulation of cyclin D1, cyclin A and cyclin B1
